| Basolateral amygdala neuropeptide Y system modulates binge ethanol consumption |
|
| Orexinergic lateral hypothalamus (LH) projections to medial septum (MS) modulate ethanol-induced sedation in male and female mice and binge-like ethanol drinking in male mice only |
|
| Chemogenetic inhibition of locus coeruleus to rostromedial tegmental nucleus noradrenergic pathway increases light cycle ethanol drinking in male and female mice and blunts ethanol-induced CTA |
|
| Somatostatin signaling modulates binge drinking behavior via the central nucleus of the amygdala |
|
| Astrocyte expression in the extended amygdala of C57BL/6J mice is sex-dependently affected by chronic intermittent and binge-like ethanol exposure |
|
| Chronic ethanol consumption exacerbates future stress‐enhanced fear learning, an effect mediated by dorsal hippocampal astrocytes |
|
| Neurocircuitry modulating drug and alcohol abuse: A preface |
|
| Chemogenetic Inhibition of Corticotropin-Releasing Factor Neurons in the Central Amygdala Alters Binge-Like Ethanol Consumption in Male Mice |
|
| Lateral habenula-projecting central amygdala circuits expressing GABA and NPY Y1 receptor modulate binge-like ethanol intake in mice |
|
| Lateral hypothalamus-projecting noradrenergic locus coeruleus pathway modulates binge-like ethanol drinking in male and female TH-ires-cre mice |
|
| The paraventricular thalamus provides a polysynaptic brake on limbic CRF neurons to sex-dependently blunt binge alcohol drinking and avoidance behavior in mice |
|
| Activation of locus coeruleus to rostromedial tegmental nucleus (RMTg) noradrenergic pathway blunts binge-like ethanol drinking and induces aversive responses in mice |
|
| A role for the neuropeptide somatostatin in the neurobiology of behaviors associated with substances abuse and affective disorders |
|
| The melanocortin system as a potential target for treating alcohol use disorders: A review of pre-clinical data |
|
| A preliminary, open-label study of naltrexone and bupropion combination therapy for treating binge drinking in human subjects |
|
| Distinct and Overlapping Patterns of Acute Ethanol-Induced C-Fos Activation in Two Inbred Replicate Lines of Mice Selected for Drinking to High Blood Ethanol Concentrations |
|
| Bupropion, Alone and in Combination with Naltrexone, Blunts Binge-Like Ethanol Drinking and Intake Following Chronic Intermittent Access to Ethanol in Male C57BL/6J Mice |
|
| Characterization of the Hippocampal Neuroimmune Response to Binge-Like Ethanol Consumption in the Drinking in the Dark Model |
|
| Corticotropin Releasing Factor Type 1 and 2 Receptor Signaling in the Medial Prefrontal Cortex Modulates Binge-Like Ethanol Consumption in C57BL/6J Mice |
|
| Medial prefrontal cortex neuropeptide Y modulates binge-like ethanol consumption in C57BL/6J mice |
|
| Assessment of depression-like behavior and anhedonia after repeated cycles of binge-like ethanol drinking in male C57BL/6J mice |
|
| Assessment of ventral tegmental area-projecting GABAergic neurons from the bed nucleus of the stria terminalis in modulating binge-like ethanol intake |
|
| Modulation of Binge-like Ethanol Consumption by IL-10 Signaling in the Basolateral Amygdala |
|
| Extended Amygdala to Ventral Tegmental Area Corticotropin-Releasing Factor Circuit Controls Binge Ethanol Intake |
|
| The Role of Orexin Signaling in the Ventral Tegmental Area and Central Amygdala in Modulating Binge-Like Ethanol Drinking Behavior |
|
| Voluntary Binge-like Ethanol Consumption Site-specifically Increases c-Fos Immunoexpression in Male C57BL6/J Mice |
|
| Preface |
|
| Nucleus accumbens MC4-R stimulation reduces food and ethanol intake in adult rats regardless of binge-like ethanol exposure during adolescence |
|
| Serotonin engages an anxiety and fear-promoting circuit in the extended amygdala |
|
| Lateral hypothalamic melanocortin receptor signaling modulates binge-like ethanol drinking in C57BL/6J mice |
|
| Lateral hypothalamus GABAergic neurons modulate consummatory behaviors regardless of the caloric content or biological relevance of the consumed stimuli |
|
| IL-1 receptor signaling in the basolateral amygdala modulates binge-like ethanol consumption in male C57BL/6J mice |
|
| Evidence that Melanocortin Receptor Agonist Melanotan-II Synergistically Augments the Ability of Naltrexone to Blunt Binge-Like Ethanol Intake in Male C57BL/6J Mice |
|
| Assessment of the Effects of 6 Standard Rodent Diets on Binge-Like and Voluntary Ethanol Consumption in Male C57BL/6J Mice |
|
| NPY signaling inhibits extended amygdala CRF neurons to suppress binge alcohol drinking |
|
| Binge-Like Consumption of Ethanol and Other Salient Reinforcers is Blocked by Orexin-1 Receptor Inhibition and Leads to a Reduction of Hypothalamic Orexin Immunoreactivity |
|
| The protective effects of the melanocortin receptor (MCR) agonist, melanotan-II (MTII), against binge-like ethanol drinking are facilitated by deletion of the MC3 receptor in mice |
|
| Altered GABA(A) Receptor Expression and Seizure Threshold Following Acute Ethanol Challenge in Mice Lacking the RII beta Subunit of PKA |
|
| Histone acetylation in the nucleus accumbens shell modulates ethanol-induced locomotor activity in DBA/2J mice |
|
| "Drinking in the dark" (DID) procedures: A model of binge-like ethanol drinking in non-dependent mice |
|
| Repeated Binge-like ethanol drinking alters ethanol drinking patterns and depresses striatal GABAergic transmission |
|
| "Drinking in the dark" (DID): A simple mouse model of binge-like alcohol intake |
|
| Targeting central melanocortin receptors: A promising novel approach for treating alcohol abuse disorders |
|
| Decreased Immunoreactivity of the Polypeptide Precursor Pro-Opiomelanocortin (POMC) and the Prohormone Convertase PC1/3 After Chronic Ethanol Exposure in Sprague-Dawley Rats |
|
| MC4-R signaling within the nucleus accumbens shell, but not the lateral hypothalamus, modulates ethanol palatability in rats |
|
| Effects of sex and deletion of neuropeptide Y2 receptors from gabaergic neurons on affective and alcohol drinking behaviors in mice |
|
| Repeated cycles of binge-like ethanol (EtOH)-drinking in male C57BL/6J mice augments subsequent voluntary etoh intake but not other dependence-like phenotypes |
|
| Adolescent binge-like ethanol exposure reduces basal ?-MSH expression in the hypothalamus and the amygdala of adult rats |
|
| Control of food intake by MC4-R signaling in the lateral hypothalamus, nucleus accumbens shell and ventral tegmental area: Interactions with ethanol |
|
| The neurobiology of binge-like ethanol drinking: Evidence from rodent models |
|
| Urocortins: CRF's siblings and their potential role in anxiety, depression and alcohol drinking behavior |
|
| Central neuropeptide y modulates binge-like ethanol drinking in C57BL/6J mice via Y1 and Y2 receptors |
|
| Corticotropin releasing factor signaling in the central amygdala is recruited during binge-like ethanol consumption in C57BL/6J mice |
|
| Neuropeptide y signaling modulates the expression of ethanol-induced behavioral sensitization in mice |
|
| Assessment of voluntary ethanol consumption and the effects of a melanocortin (MC) receptor agonist on ethanol intake in mutant C57BL/6J mice lacking the MC-4 receptor |
|
| CRF-1 antagonist and CRF-2 agonist decrease binge-like ethanol drinking in c57bl/6j mice independent of the HPA axis |
|
| Ethanol-induced increase of agouti-related protein (AgRP) immunoreactivity in the arcuate nucleus of the hypothalamus of C57BL/6J, but not 129/SvJ, inbred mice |
|
| Neuropeptide y conjugated to saporin alters anxiety-like behavior when injected into the central nucleus of the amygdala or basomedial hypothalamus in BALB/cJ mice |
|
| Pre-clinical evidence that Corticotropin-Releasing Factor (CRF) receptor antagonists are promising targets for pharmacological treatment of alcoholism |
|
| Deletion of agouti-related protein blunts ethanol self-administration and binge-like drinking in mice |
|
| The alcohol deprivation effect in C57BL/6J mice is observed using operant self-administration procedures and is modulated by CRF-1 receptor signaling |
|
| Blockade of the corticotropin releasing factor type 1 receptor attenuates elevated ethanol drinking associated with drinking in the dark procedures |
|
| Decreased immunoreactivity of the melanocortin neuropeptide ?-melanocyte-stimulating hormone (?-MSH) after chronic ethanol exposure in Sprague-Dawley rats |
|
| The CRF-1 receptor antagonist, CP-154,526, attenuates stress-induced increases in ethanol consumption by BALB/cJ mice |
|
| Effects of food availability and administration of orexigenic and anorectic agents on elevated ethanol drinking associated with drinking in the dark procedures |
|
| Corticotropin releasing factor-1 receptor antagonist, CP-154,526, blocks the expression of ethanol-induced behavioral sensitization in DBA/2J mice |
|
| Elevated anxiety-like behavior following ethanol exposure in mutant mice lacking neuropeptide Y (NPY) |
|
| Involvement of protein kinase A in ethanol-induced locomotor activity and sensitization |
|
| Increased consumption but not operant self-administration of ethanol in mice lacking the RII? subunit of protein kinase A |
|
| Effects of melanocortin receptor activation and blockade on ethanol intake: A possible role for the melanocortin-4 receptor |
|
| Comparison of basal neuropeptide Y and corticotropin releasing factor levels between the high ethanol drinking C57BL/6J and low ethanol drinking DBA/2J inbred mouse strains |
|
| Reduced anorexigenic efficacy of leptin, but not of the melanocortin receptor agonist melanotan-II, predicts diet-induced obesity in rats |
|
| Peripheral and central administration of a selective neuropeptide Y Y1 receptor antagonist suppresses ethanol intake by C57BL/6J mice |
|
| A role for neuropeptide Y in neurobiological responses to ethanol and drugs of abuse |
|
| Assessment of ethanol consumption and water drinking by NPY Y2 receptor knockout mice |
|
| Predictors of high ethanol consumption in RII? knock-out mice: Assessment of anxiety and ethanol-induced sedation |
|
| Conditioned taste aversion memory and c-Fos induction are disrupted in RII?-protein kinase A mutant mice |
|
| Nitrous oxide-induced c-Fos expression in the rat brain |
|
| MTII-induced reduction of voluntary ethanol drinking is blocked by pretreatment with AgRP-(83-132) |
|
| Alcoholism and obesity: Overlapping neuropeptide pathways? |
|
| Central neuropeptide Y alters ethanol-induced sedation,but not ethanol intake, in C57BL/6 mice |
|
| Induction of a brainstem correlate of conditioned taste aversion expression: Role of the pontine parabrachial nucleus |
|
| Voluntary alcohol consumption is controlled via the neuropeptide Y Y1 receptor. |
|
| Inhibition of protein kinase A activity interferes with long-term, but not short-term, memory of conditioned taste aversions |
|
| Dopamine mediation of the feeding response to violations of spatial and temporal expectancies |
|
| Comparison of central administration of corticotropin-releasing hormone and urocortin on food intake, conditioned taste aversion, and c-Fos expression |
|
| cFos induction during conditioned taste aversion expression varies with aversion strength |
|
| Neurobiological responses to ethanol in mutant mice lacking neuropeptide Y or the Y5 receptor |
|
| High ethanol consumption and low sensitivity to ethanol-induced sedation in protein kinase A-mutant mice. |
|
| Ethanol-induced c-Fos expression in catecholamine- and neuropeptide Y- producing neurons in rat brainstem |
|
| The induction of c-Fos in the NTS after taste aversion learning is not correlated with measures of conditioned fear |
|
| Insular cortex lesions and taste aversion learning: Effects of conditioning method and timing of lesion |
|
| Metabolic, gastrointestinal, and CNS neuropeptide effects of brain leptin administration in the rat |
|
| Glucagon-like peptide-1 (7-36) amide: A central regulator of satiety and interoceptive stress |
|
| Effects of third intracerebroventricular injections of corticotropin-releasing factor (CRF) on ethanol drinking and food intake |
|
| Central infusion of glucagon-like peptide-1-(7-36) amide (GLP-1) receptor antagonist attenuates lithium chloride-induced c-Fos induction in rat brainstem |
|
| Effects of food deprivation on conditioned taste aversions in rats |
|
| Learned tolerance to Ethanol-induced c-Fos expression in rats |
|
| Conditioning method dramatically alters the role of amygdala in taste aversion learning |
|
| Ethanol consumption and resistance are inversely related to neuropeptide Y levels |
|
| Ethanol consumption and resistance are inversely relted to neuropeptide Y levels |
|
| Ethanol consumption in rats selectively bred for differential saccharin intake |
|
| Central infusion of melanocortin agonist MTII in rats: Assessment of c- Fos expression and taste aversion |
|
| Dopamine and sodium appetite: Antagonists suppress sham drinking of NaCl solutions in the rat |
|
| Ethanol-induced c-Fos expression in rat lines selected for low and high alcohol consumption |
|
| Central infusion of GLP-1, but not leptin, produces conditioned taste aversions in rats |
|
| Central leptin stimulates corticosterone secretion at the onset of the dark phase |
|
| Melanocortin receptors in leptin effects [7] |
|
| Continuous intraoral saccharin infusions in high vs. low saccharin-consuming rats |
|
| Glucagon-like peptide-1 and satiety [5] (multiple letters) |
|
| Taste reactivity and consumption measures in the assessment of overshadowing: Modulation of aversive, but not ingestive, reactivity |
|
| Central infusions of leptin and GLP-1-(7-36) amide differentially stimulate c-FLI in the rat brain |
|
| Delayed generalization testing produces enhanced alcohol aversions in rats |
|
| c-Fos induction in rat brainstem in response to ethanol- and lithium chloride-induced conditioned taste aversions |
|
| Taste-potentiated color aversions in pigeons: Examination of CS preexposure and subsequent effects on postconditioning extinction of the taste aversion |
|
