Neurobehavioral and emotional alterations in a preclinical model of compulsivity. |
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Non-invasive transcranial brain stimulation (NIBS) for motor and cognitive recovery in animal models of stroke: A systematic review. |
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Rodent version of the Iowa Gambling Task: Individual differences in risk decision-making behavior. Preclinical evicences |
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Presentation of initiative women in Neuroscience Wins Talk |
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Chemogenetic orbito frontal cortex and amygdala circuit activation and inhibition in high compulsive rats |
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Efficacy of two types of Transcranial direct-current stimulation on frontal cortex in a preclinical model of compulsivity |
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Is there any effects in risky decision making behavior after a ischemic stroke? Preclinical evidences |
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Is there any influence of compulsivity on decision-making behavior? Preclinical evidence |
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Impulsive behavior assessment in a preclinical model of stroke |
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The effects in risky decision making in a preclinical model of ischemic stroke. |
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Individual differences in risk decision-making strategies: an study using a rat gambling task. |
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Altered neuroanatomical mechanisms of a compulsive phenotype selected by Schedule-Induced Polydipsia |
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A chemogenetic approach to disentangle the brain circuit function in high compulsive rats. |
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Effects Transcranial direct-current stimulation on frontal cortex in a preclinical model of compulsivity |
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Chemogenetic orbito frontal cortex inhibition and chemogenetic amygdala activation in high compulsive rats |
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Effects of Transcranial direct-current stimulation on medial prefrontal cortex in a preclinical model of compulsivity |
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Neurostructural abnormalities in High Drinkers compulsive rats selected by Schedule-Induced Polydipsia. |
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Cognitive inflexibility mediated by memory impairment and decreased frontal gene expression of HTR2A, Grin1, and BDNF in a preclinical model of compulsivity. |
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Altered neurocognitive and emotional mechanisms in a preclinical model of compulsivity. |
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High-fat diet induces long-term vulnerability to impulsive behavior: preclinical studies. |
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Emotional deficit and stress regulation in high compulsive rats selected by Schedule-Induced Polydipsia |
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Increased impulsivity by chronic high-fat diet: evidences in a preclinical study. |
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Reduced cortical 5HTR2A gene expression in high compulsive drinker rats. |
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How uncertainty affects decision-making in a compulsive phenotype of rats selected by Schedule-Induced Polydipsia |
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Impaired decision-making and social regulation in a compulsive phenotype of rats selected by Schedule-Induced Polydipsia |
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Metabolomic alterations underlying compulsive behavior in a preclinical model |
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Resistant emotional memory and blunted HPA axis response stress in a compulsive phenotype of rats |
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The effect of high-fat diet on impulsivity: lessons from a preclinical model |
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Altered memory retrieval and frontal gene expression of HTR2A, Grin1, and BDNF in a preclinical model of compulsivity |
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Resistant emotional memory and blunted neuroendocrine response of HPA axis in a compulsive phenotype of rats |
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Altered memory retrieval and NMDA and 5-HT2A gene receptors in high compulsive rats |
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A preclinical model of individual differences in social-reward learning |
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Preclinical study on a common trait of different neuropsychiatric disorders: Compulsivity. |
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Glutamatergic modulators as a new tool to treat compulsivity: preclinical studies on schedule-induced polydipsia. |
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Compulsivity, a common trait in different neuropsychiatric disorders: preclinical studies on schedule-induced polydipsia. |
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Glutamatergic modulators as a new tool to treat compulsivity: preclinical studies on schedule-induced polydipsia |
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Glutamatergic drugs in compulsivity: preclinical studies in schedule-induced polydipsia |
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Long-term increased impulsive response after group A streptococcal antigen and antibiotic exposition in adolescent rats |
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Stress and immune activation: effects on inhibitory control deficit |
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Do psychoactive drugs play a therapeutic role in compulsivity? |
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Potential therapeutic effect of psychoactive drugs in compulsivity |
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Inhibitory control deficit in rats: possible contribution of immune activation and stress factors. |
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