| Positive allosteric γ‐aminobutyric acid type A receptor modulation prevents lipotoxicity‐induced injury in hepatocytes in vitro |
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| DPP4 and ACE2 in Diabetes and COVID-19: Therapeutic Targets for Cardiovascular Complications? |
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| DPP4 deletion in adipose tissue improves hepatic insulin sensitivity in diet-induced obesity |
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| Visfatin/eNampt induces endothelial dysfunction in vivo: a role for Toll-Like Receptor 4 and NLRP3 inflammasome |
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| Soluble dipeptidyl peptidase 4 triggers endothelial cell senescence: which role for thromboxane A2 (TXA2)? |
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| The angiotensin-(1-7)/Mas receptor axis protects from endothelial cell senescence via klotho and Nrf2 activation |
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| SOLUBLE DIPEPTIDYL PEPTIDASE 4 INDUCES SENESCENCE IN HUMAN ENDOTHELIAL CELLS |
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| THE PHARMACOLOGICAL BLOCKADE OF INTERLEUKIN-1 beta PREVENTS ENDOTHELIAL SENESCENCE AND VASCULAR SMOOTH MUSCLE CELL INFLAMMATION |
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| IS VISFATIN INVOLVED IN THE VASCULAR DYSFUNCTION ASSOCIATED WITH DIABESITY AND AGEING? |
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| ANGIOTENSIN-(1-7) ATTENUATES ENDOTHELIAL CELL SENESCENCE VIA NRF2 ACTIVATION |
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| THE ANGIOTENSIN-(1-7)/ MAS RECEPTOR AXIS COUNTERACT PRO-INFLAMMATORY SIGNALING IN HUMAN VASCULAR SMOOTH MUSCLE CELLS |
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| DPP4 DELETION IN ADIPOSE TISSUE PROMOTES HEPATIC INSULIN SENSITIVITY VIA IGF1 |
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| IGF1 Links DPP-4 to Hepatic Insulin Sensitivity |
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| Inflammation, glucose, and vascular cell damage: the role of the pentose phosphate pathway (vol 15, 82, 2016) |
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| Protease-Activated Receptor 2 Promotes Pro-Atherogenic Effects through Transactivation of the VEGF Receptor 2 in Human Vascular Smooth Muscle Cells |
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| Soluble Dipeptidyl Peptidase-4 Impairs Vasodilatation via Proteinase-Activated Receptor-2 and Thromboxane A2 |
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| The angiotensin-(1-7)/mas axis counteracts angiotensin II-dependent and -independent pro-inflammatory signaling in human vascular smooth muscle cells |
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| Eicosapentaenoic acid and arachidonic acid differentially regulate adipogenesis, acquisition of a brite phenotype and mitochondrial function in primary human adipocytes |
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| Inflammation, glucose, and vascular cell damage: the role of the pentose phosphate pathway |
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| Soluble dipeptidyl peptidase-4 induces microvascular endothelial dysfunction through proteinase-activated receptor-2 and thromboxane A(2) release |
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| ANGIOTENSIN-(1-7) MITIGATES VASCULAR INFLAMMATION AND SENESCENCE THROUGH MAS RECEPTOR |
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| THE ADIPOKINE SOLUBLE DIPEPTIDYL PEPTIDASE-4 IMPAIRS MICROVASCULAR REACTIVITY THROUGH PROTEINASE-ACTIVATED RECEPTOR-2 AND THROMBOXANE A2 PRODUCTION |
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| PAR2 is upregulated by adipocyte-derived factors and high fat diet in the vascular wall |
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| Adipose-specific Dipeptidyl Peptidase 4 (DPP4) knockout mice display improved fasting insulin and smaller adipocytes on High Fat Diet (HFD) |
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| Eicosapentaenoic Acid but Not Docosahexaenoic Acid Promotes a Brite Phenotype in Primary Human Adipocytes |
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| Adipose-specific Dipeptidyl Peptidase-4 (DPP-4) Knockout Mice Display Improved Fasting Insulin and Cholesterol Levels Despite Increased Weight Gain on HFD |
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| Adipose-Tissue Specific Deletion of Dipeptidyl Peptidase-4 (DPP-4) Enhances M2 Macrophage Markers and Results in Smaller Adipocytes Under HFD |
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| Nutritional ingredients modulate adipokine secretion and inflammation in human primary adipocytes |
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| The interleukin-1 receptor antagonist anakinra improves endothelial dysfunction in streptozotocin-induced diabetic rats |
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| Soluble Dipeptidyl Peptidase-4 Induces Endothelial Dysfunction by the Release of Vasoconstrictor Prostanoids: Protective Effect of Dipeptidyl Peptidase Inhibitors |
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| Effect of the n-3 LC-PUFA EPA on white-to-brown transition of primary human adipose-derived stem cells |
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| Inflammatory stimulation transforms glucose into a deleterious agent in human vascular smooth muscle cells |
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| Soluble dipeptidyl peptidase 4 induces inflammation and proliferation of human vascular smooth muscle cells via protease-activated receptor 2 |
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| MICROVASCULAR ENDOTHELIAL DYSFUNCTION BY SOLUBLE DIPEPTIDYL PEPTIDASE-4: RELEASE OF THROMBOXANE-A(2) |
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| Inflammation Converts Glucose Into A Deleterious Agent In Human Aortic Smooth Muscle Cells |
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| Soluble DPP4 induces inflammation and proliferation of human smooth muscle cells via protease-activated receptor 2 |
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| THE ANGIOTENSIN (1-7)/MAS RECEPTOR AXIS AND VASCULAR CELL INFLAMMATION |
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| THE ADIPOKINE VISFATIN/NAMPT INDUCES PREMATURE SENESCENCE AND TELOMERE DAMAGE IN HUMAN ENDOTHELIAL CELLS |
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| Visfatin/Nampt induces telomere damage and senescence in human endothelial cells |
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| THE ADIPOKINE VISFATIN/NAMPT PROMOTES TELOMERE DAMAGE AND PREMATURE SENESCENCE IN HUMAN ENDOTHELIAL CELLS |
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| THE ACTIVATION OF THE ANGIOTENSIN (1-7)/MAS RECEPTOR AXIS PROTECTS AGAINST VASCULAR CELL INFLAMMATION |
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| SOLUBLE DIPEPTIDYL PEPTIDASE-4 IMPAIRS MICROVASCULAR ENDOTHELIUM-DEPENDENT RELAXATION: A ROLE FOR VASOCONSTRICTOR PROSTANOIDS |
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| DPP-4 Is a Novel Adipokine Contributing to Endothelial Dysfunction Through Its Enzymatic Activity and the Release of Thromboxane A2 |
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| Adipose tissue and its role in organ crosstalk |
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| BMP4 and BMP7 induce the white-to-brown transition of primary human adipose stem cells |
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| Visfatin/Nampt: An Adipokine with Cardiovascular Impact |
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| Evidence against a beneficial effect of irisin in humans. |
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| Visfatin as a Novel Mediator Released by Inflamed Human Endothelial Cells |
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| THE ADIPOKINE VISFATIN INDUCES TELOMERE DAMAGE AND CELL SENESCENCE IN HUMAN ENDOTHELIAL CELLS |
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| The novel adipokines BMP4 and BMP7 promote browning of primary human adipose-derived stem cells |
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| Endothelial dysfunction induced by dipeptidyl peptidase-4 in isolated mice mesenteric microvessels |
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| Evidence against a beneficial effect of irisin in humans |
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| Evidence against a Beneficial Effect of Irisin in Humans |
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| Dipeptidyl Peptidase-4 Impairs Microvascular Endothelial-Dependent Relaxation: The Role of Cyclooxygenase |
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| The Novel Adipokines BMP4 and BMP7 Induce Browning in Primary Human Adipose Stem Cells |
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| Inflammation enhances visfatin synthesis in the vascular wall |
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| Evidence against a beneficial effect of irisin in humans |
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| Pathways responsible for apoptosis resulting from Amadori-induced oxidative and nitrosative stress in human mesothelial cells |
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| VISFATIN IMPAIRS ENDOTHELIUM-DEPENDENT RELAXATION IN HUMAN PENILE RESISTANCE ARTERIES THROUGH NICOTINAMIDE PHOPHORIBOSYL TRANSFERASE (NAMPT) ACTIVITY |
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| Visfatin Impairs Endothelium-Dependent Relaxation in Rat and Human Mesenteric Microvessels through Nicotinamide Phosphoribosyltransferase Activity |
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| VISFATIN IMPAIRS ENDOTHELIUM-DEPENDENT RELAXATION IN MICROVESSELS: ROLE OF NAMPT AND NAD |
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| THE ANGIOTENSIN-(1-7)/MAS AXIS EXHIBITS ANTI-INFLAMMATORY PROPERTIES IN VASCULAR HUMAN SMOOTH MUSCLE CELLS |
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| Inhibition of vascular endothelial growth factor (VEGF)-induced endothelial proliferation, arterial relaxation, vascular permeability and angiogenesis by dobesilate |
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| Visfatin/PBEF/Nannpt: a new cardiovascular target? |
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| Inflammation Determines the Pro-Adhesive Properties of High Extracellular D-Glucose in Human Endothelial Cells In Vitro and Rat Microvessels In Vivo |
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| Extracellular PBEF/NAMPT/visfatin activates pro-inflammatory signalling in human vascular smooth muscle cells through nicotinamide phosphoribosyltransferase activity |
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| VISFATIN/NAMPT/PBEF: ROLE IN VASCULAR WALL INFLAMMATION |
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| THE ADIPOKINE VISFATIN IS SYNTHESIZED BY HUMAN ENDOTHELIAL CELLS AND EXERTS A DIRECT INFLAMMATORY EFFECT ON HUMAN SMOOTH MUSCLE CELLS. |
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| Insulin Resistance, Endothelial Dysfunction and Inflammation: A Role for Adipokines |
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| The adipokine visfatin is synthesized by human endothelial cells and promotes inflammation in human smooth muscle cells |
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| In vitro and In vivo induction of adhesion molecules and leukocyte recruitment: interaction between high glucose and inflammation |
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| The deleterious effect of high concentrations of D-glucose requires pro-inflammatory preconditioning |
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